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Gene Variant Raises Heart Failure Risk After Myocarditis in Children — girl in blue jacket holding red and silver ring

Gene Variant Raises Heart Failure Risk After Myocarditis in ChildrenCEFR A2

6 Dec 2025

Adapted from Unknown author, Futurity CC BY 4.0

Photo by Patty Brito, Unsplash

AI-assisted adaptation of the original article, simplified for language learners.

A new study found that 34.4% of children who developed dilated cardiomyopathy after myocarditis had a cardiomyopathy gene variant, while only 6.3% of control children had such variants. The authors say the difference was highly significant.

The research compared 32 children with both dilated cardiomyopathy and myocarditis to children who had myocarditis without dilated cardiomyopathy and to heart-healthy controls. The children with cardiomyopathy were part of the Pediatric Cardiomyopathy Registry (PCMR). Steven E. Lipshultz founded and leads the PCMR. Researchers describe a "double hit" model: a genetic mutation from birth plus an infection that reaches heart cells. The authors advise genetic testing and say the work appears in Circulation Heart Failure.

Difficult words

  • dilated cardiomyopathyA heart condition with a weak, enlarged heart.
  • myocarditisInflammation or infection of the heart muscle.
  • variantA different form of a gene.
  • significantAn important result not likely by chance.
  • registryA recorded list of people or cases.
  • double hitTwo causes that together make disease happen.
  • mutationA change in a person's genes present at birth.
  • genetic testingA medical test that looks for gene changes.

Tip: hover, focus or tap highlighted words in the article to see quick definitions while you read or listen.

Discussion questions

  • What does the "double hit" idea mean in this article?
  • How could genetic testing help families with a child who has heart problems?
  • Do you think it is important to compare sick children with healthy controls? Why or why not?

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